Taint length correlated to male fertility

From Geekologie
(Taint That The Truth!: Length of A Man's Gooch Directly Related To His Fertility)

" According to a recent study, the length of a man's taint is directly related to his fertility, with shorter measurements having a significantly greater chance of being infertile."

Reference
Mendiola J, Stahlhut RW, Jørgensen N, Liu F, Swan SH (2011) Shorter Anogenital Distance Predicts Poorer Semen Quality in Young Men in Rochester, New York Environmental Health Perspectives-. doi:10.1289/ehp.1103421

Abstract
Background: In male rodents, anogenital distance (AGD) provides a sensitive and continuous correlate of androgen exposure in the intrauterine environment and predicts later reproductive success. Some endocrine disrupting chemicals can alter male reproductive tract development, including shortening AGD, in both rodents and humans. Whether AGD is related to semen quality in human is unknown.

Objectives: To examine associations between AGD and semen parameters in adult males.

Methods: We used multiple regression analyses to model the relationships between sperm parameters and two alternative measures of AGD (anus to the posterior base of the scrotum [AGDAS], and to the cephalad insertion of the penis [AGDAP]), in 126 volunteers in Rochester, NY.

Results: AGDAS, but not AGDAP, was associated with sperm concentration, motility, morphology, total sperm count and total motile count (p-values 0.002-0.048). Men with AGDAS below (compared to above) the median were 7.3 times more likely (95% CI 2.5, 21.6) to have a low sperm concentration (<20x106/ml). For a typical study participant, sperm concentrations were 34.7 x106/ml and 51.6 x106/ml at the 25th and 75th percentiles of (adjusted) AGDAS. Conclusions: In our population, AGDAS was a strong correlate of all semen parameters and a predictor of low sperm concentration. In animals, male AGD at birth reflects androgen levels during the masculinization programming window and predicts adult AGD and reproductive function. Our results suggest, therefore, that the androgenic environment during early fetal life exerts a fundamental influence on both AGD and adult sperm counts in humans, as demonstrated in rodents.